Fibrinolysis after Heart Transplantation

Thromb Haemost 1988; 59(01): 123
DOI: 10.1055/s-0038-1642579

Review Article

Schattauer GmbH Stuttgart

Authors

J A Páramo

Haematology Services, University Clinic. University of Navarra, Pamplona, Spain
R Arcas

^*Cardiovascular Services, University Clinic. University of Navarra, Pamplona, Spain
J Herreros

^*Cardiovascular Services, University Clinic. University of Navarra, Pamplona, Spain
B Cuesta

Haematology Services, University Clinic. University of Navarra, Pamplona, Spain
R Llorens

^*Cardiovascular Services, University Clinic. University of Navarra, Pamplona, Spain
E Rocha

Haematology Services, University Clinic. University of Navarra, Pamplona, Spain

References
1 Knight M TN, Dawson R, Melrose DG. Fibrinolytic response to surgery. Labile and stable patterns and their relevance to postoperative deep vein thrombosis. Lancet 1977; 1: 370-3
2 Stibbe J, Kluft C, Brommer E JP et al. Enhanced fibrinolytic activity during cardiopulmonary bypass in open-heart surgery in man is caused by extrinsic (tissue-type) plasminogen activator. Eur J Clin Invest 1984; 14: 375-82
3 Mammen EF, Koets MH, Washington BC et al. Hemostasis changes during cardiopulmonary bypass surgery. Sem Thromb Hemostas 1985; 11: 281-92
4 D’Angelo A, Kluft C, Verheijen JH, Rijken DC, Mozzi E, Mannucci PM. Fibrinolytic shut-down after surgery: impairment of the balance between tissue-type plasminogen activator and its specific inhibitor. Eur J Clin Invest 1985; 15: 308-12
5 Páramo JA, Campbell W, Cuesta B, Gómez C, Aranda A, Rocha E. Fibrinolytic response in malignancy. Fibrinolysis 1987; 1: 195-9