Thorac Cardiovasc Surg 1981; 29(6): 329-336
DOI: 10.1055/s-2007-1023509
© Georg Thieme Verlag Stuttgart · New York

Conduit Repair for Complex Congenital Heart Disease with Pulmonary Atresia or Right Ventricular Outflow Tract Obstruction*,**

Part I: Surgical ResultsE. R. de Vivie, K. Hellberg, D. D. Kutzner, G. Rahlf1 , G. Rupprath2 , A. J. Beuren2
  • Center of Thoracic and Cardiovascular Surgery,
  • 1Department of Pathology,
  • 2Department of Pediatric Cardiology, University of Göttingen
*Dedicated to Prof. Dr. J. Koncz on the occasion of his 65th birthday**Supported by Deutsche Forschungsgemeinschaft, SFB 89, Kardiologie Göttingen
Further Information

Publication History

1981

Publication Date:
19 March 2008 (online)

Summary

From 1972 to 1981, 40 valved extracardiac conduits have been implanted to bridge the right ventricular outflow tract (RVOT) in 37 patients for different complex congenital cardiac anomalies. The patients ranged in age from 2 to 23 years (mean 8.5 years). Two Dacron prostheses with incorporated Lillehei-Kaster valves, 1 valveless Dacron conduit, 27 Hancock conduits, and 9 lonescu-Shiley conduits were chosen according to availability and the progress in conduit technology. Thirty-three patients had had up to 4 previous palliative procedure before conduit repair. Although the complication rate appears considerable, the mortality rate seemed to be unaffected by the high percentage of prior surgery. Exchange of the conduit became necessary in 2 children, because of conduit Stenosis with calcification of the Hancock valve 5 and 7 years respectively after Implantation, and in another patient an outgrown Lilleher-Kaster valve Dacron conduit had to be replaced 9 years after repair. Five children died in the early postoperative course: one in pulmonary failure and kinking of the conduit; one with right heart failure and pulmonary hypertension stage IV; 2 in myocardial failure, and one due to severe bleeding from the prosthesis. Three patients died in the late postoperative course 11 weeks, 4 years and 4 years respectively after conduit repair. Conduit surgery offers new possibilities for repair of complex cyanotic cardiac malformations. At present, however, none of the commercially available conduits is an ideal substitute. Our limited experience with the hemodynamic results of lonescu Shiley conduits is promising although longer observation periods are needed for a definitive jugdement. At present, the largest possible conduit should be used whenever possible; Problems of compression or kinking can be avoided with proper positioning of the conduit.

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