Abstract
Increases in cadence may augment SV during submaximal cycling (> 65 % V·O2max) via effects of increased muscle pump activity on preload. At lower workloads (45
- 65 % V·O2max), SV tends to plateau, suggesting that effects of increases in cadence on pump activity
have little influence on SV. We hypothesized that cadence-induced increases in CO
at submaximal workloads, where SV tends to plateau, are due to elevations in HR and/or
O2 extraction. SV, CO, HR, V·O2, and Δa - vO2 were assessed at 80 and 100 rpm during workloads of 50 % (LO) or 65 % (HI) of V·O2max in 11 male cyclists. No changes in SV were seen. CO was higher at 100 rpm in 10 of
11 subjects at LO (18.1 ± 2.7 vs. 17.2 ± 2.6 L/min). V·O2 at both workloads was greater at 100 than 80 rpm as was HR (LO: 129 ± 11 vs. 121
± 10 beats/min; HI: 146 ± 13 vs. 139 ± 14 beats/min) (p < 0.05). Δa - vO2 was greater at HI compared to LO at 80 (15.1 ± 1.6 vs. 13.6 ± 1.3 ml) and 100 rpm
(16.0 ± 1.7 vs. 15.1 ± 1.6 ml) (p < 0.05). Results suggest that increases in O2 demand during low submaximal cycling (50 % V·O2max) at high cadences are met by HR-induced increases in CO. At higher workloads (65
% V·O2max), inability of higher cadences to increase CO and O2 delivery is offset by greater O2 extraction.
Key words
cycling - cadence - cardiac output - stroke volume - heart rate - arterial‐venous
oxygen difference
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Dr. Ph.D. Charles L. Stebbins
Internal Medicine, Division of Cardiovascular Medicine
University of California, Davis
One Shields Ave. TB - 172
95616 Davis, California
United States
eMail: clstebbins@ucdavis.edu