Semin Liver Dis 2023; 43(03): e2
DOI: 10.1055/s-0043-1776036
Erratum

Erratum to: Genetic Contributions to Biliary Atresia: A Developmental Cholangiopathy

1   Division of Pediatric Gastroenterology, Hepatology, and Nutrition, Children's Healthcare of Atlanta and Emory University School of Medicine, Atlanta, Georgia
,
1   Division of Pediatric Gastroenterology, Hepatology, and Nutrition, Children's Healthcare of Atlanta and Emory University School of Medicine, Atlanta, Georgia
› Author Affiliations

The authors have informed the publisher that in this article (with DOI 10.1055/a-2153-8927; published online on September 19, 2023), [Tables 3] and [4] have been swapped. The correct [Table 3] and [Table 4] are as follows:

Table 3

Biliary atresia-associated microRNAs and their hypothesized target genes

miRNA

Target

Analyses

miR-19b

TGFßR2

140

miR-21

RASGRP1 (upstream of Erk/Mek), PTEN

141,142

miR-29b,c

DNMT3A, DNMT3B, IFN-γ

115,136,143

miR-140–3p

FGF9

144

miR-142–5p

IFN-γ

136

miR-145

ADD3

37

miR-155

SOCS1, IFN-γ

145,146

miR-200b

FOG2

116,144

miR-214

SUFU

147

miR-499

SOX6, ETS1

148,149

miR-4429

MAPK1, AKT3, PTEN

150

miR-4689

SGK1

150

Table 4

Hypomethylated regions identified in biliary atresia patients

Hypomethylated gene (or region)

Cell type

Analyses

SHH, GLI2, PDGFA

Hepatocytes, cholangiocytes

101

Alu, LINE-1, SAT2 (retrotransposons)

Peripheral blood leukocytes

118

IFN-γ promoter

Hepatocytes, cholangiocytes, CD4+ T cells

136,137

ATX promoter

Peripheral blood leukocytes

138

CD11a promoter

CD4+ T cells

120

FOXP3 promoter

Treg cells

139



Publication History

Article published online:
12 October 2023

© 2023. Thieme. All rights reserved.

Thieme Medical Publishers, Inc.
333 Seventh Avenue, 18th Floor, New York, NY 10001, USA