Rebuttal: The independent association of nitric oxide synthase 3 polymorphisms and their combination with inflammatory markers

Thromb Haemost 2007; 97(04): 683-684
DOI: 10.1160/TH07-02-0101

Letters to the Editor

Schattauer GmbH

Authors

Ignatios Ikonomidis

¹2^nd Department of Cardiology, Athens University, Athens, Greece
John Lekakis

¹2^nd Department of Cardiology, Athens University, Athens, Greece
Maria Tsibida

¹2^nd Department of Cardiology, Athens University, Athens, Greece
Dimitrios T. Kremastinos

¹2^nd Department of Cardiology, Athens University, Athens, Greece

References
1 Fergusson W. @@@Nitric oxide synthase 3 genotypes and inflammatory markers in coronary artery disease patients. Thromb Haemost 2007; 97: 683
2 Nagassaki S, Sertorio JTC, Metzger IF. et al. eNOS gene T-786C polymorphism modulates atorvastatin-induced increase in blood nitrite. Free Radic Biol Med 2006; 41: 1044-1049.
3 Souza-Costa DC, Sandrim VC, Lopes LF. et al. Anti-inflammatory effects of atorvastatin: Modulation by the T-786C polymorphism in the endothelial nitric oxide synthase gene. Atherosclerosis. 2006 doi:10.1016/j.atherosclerosis.2006.07.020.
4 Lekakis JP, Ikonomidis I, Tsibida M. et al. Genetic variations of the endothelial nitric oxide synthase gene are related to increased levels of C-reactive protein and macrophage-colony stimulating-factor in patients with coronary artery disease. Thromb Haemost 2006; 96: 520-528.