Inflammation has been accepted as a key pathobiological mechanism in the development
            of cardiovascular disease, and acceptance is likely on its way for diabetes as well.
            Inflammation provides a linking mechanism between these two disorders, allowing them
            to influence each other in a wide variety of ways. Interventions aimed broadly at
            inflammation are likely to influence both diseases, and pathway-specific interventions
            may prove useful as well. The role of genetic variation remains poorly understood.
            
         
         
         
            Diabetes - insulin resistance - obesity - inflammation - thrombosis