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Thromb Haemost 2008; 100(05): 731-732
DOI: 10.1160/TH08-10-0646
DOI: 10.1160/TH08-10-0646
Editorial Focus
A radical explanation for the effect of the HPA-1b polymorphism in platelet αIIbβ3-integrin?
Further Information
Publication History
Received
07 October 2008
Accepted
07 October 2008
Publication Date:
22 November 2017 (online)
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References
- 1 Essex DW. The role of thiols and disulfides in platelet function. Antioxid Redox Signal 2004; 06: 736-746.
- 2 Weiss EJ, Bray PF, Tayback M. et al. A polymorphism of a platelet glycoprotein receptor is an inherited risk factor for coronary thrombosis. N Engl J Med 1996; 334: 1090-1094.
- 3 Vijayan KV, Bray PF. Molecular Mechanisms of prothrombotic risk due to genetic variations in platelet genes: Enhanced outside-in signaling through the Pro33 variant of integrin β3 . Exp Biol Med 2006; 231: 505-513.
- 4 Jones CI, Garner SF, Angenent W. et al. on behalf of the BLOODOMICS consortium. Mapping the platelet profile for functional genomic studies and demonstration of the effect size of the GP6 locus. J Thromb Haemost 2007; 05: 1756-1765.
- 5 Ball C, Vijayan KV, Nguyen T. et al. Glutathione regulates integrin αIIbβ3-mediated cell adhesion under flow conditions. Thromb Haemost 2008; 100: 857-863.